Introduction

Left ventricular (LV) aneurysms result from large transmural myocardial infarctions (MI).[1],[2] Historical estimates suggest aneurysms occur in up to a third of patients with Q-wave infarctions. In the contemporary era, however, the incidence is significantly lower due to improved focus on “door to balloon time” and optimal medical therapy. True LV aneurysms are made of fibrous scar tissue, a property that makes them less vulnerable to rupture yet still leads to a maladaptive process characterized by adverse ventricular remodeling and a progressive decline in LV function.